Occam's Aftershave
Posts: 5287 Joined: Feb. 2006
|
Quote (RedDot @ Aug. 01 2007,22:03) | Quote (Occam's Aftershave @ July 25 2007,00:08) | Like I said, you completely blew off the criticism about "no evolutionary predictions" and made more completely unsupported assertions that the basis of the predictions used are wrong.
That does nothing to counter the rebuttal to your claim that the medical community does not use ToE's predictive power.
BTW there are dozens of papers on PubMed using the theory to predict the evolution of pathogens. Here are but a few
Imperfect vaccines and the evolution of pathogen virulence
Epidemiology, Evolution, and Future of the HIV/AIDS Pandemic
Quote | Thanks. I'm going on vacation for a week. See you all when I return. The responses should make interesting reading. |
Hopefully you won't be another 'fart and dart' creationist who makes it a "permanent vacation". :D |
If pathogen virulence increasing in the presence of antibiotics is the best you have, then you're a long way from your stated goal. Both papers make lip service only to evolution. Neither have an actual prediction of what will happen when a particular pathogen is introduced to a particular antibody.
That being said, you are now screaming at the computer screen for proof. Ok, I'll appease you. While there might be one or two that slip through this net, almost all pathogens lose information as they become resistant to an antibiotic. Evolutionary theory specifically states that enviromental pressure will cause an organism to gain information in the way of new genes or more specific proteins. However it is just not the case. Here are some examples for you:
In the presence of: Actinonin, the phenotype (PT) displays a loss of enzyme activity
Actinonin, the PT has an SOS response halting cell division
Actinonin, the PT loses a regulatory protein.
Erythromycin, the PT has reduced affinity to 23S rRNA or loss of a regulatory protein.
Nalidixic Acid, the PT experiences loss or inactivation of a regulatory protein
and so on, and so on, and so on...
There are examples of resistance through gene transfer, but that does not explain the origin of those genes. Where mutations are oberved, these mutations result in the loss of pre-existing cellular systems/activities, such as porins and other transport systems, regulatory systems, enzyme activity, and protien binding. Such losses are never compensated, unless resistance is lost, and cannot be held up as valid examples of evolutionary change.
You guys keep giving me underhand softballs, I'll keep swinging. |
Oh jeez, the crackpot's back, and he's in full Gish Gallop mode. Do we really have to deal with the same asinine PRATT creto arguments again?
OK RedDot, please start by defining biological information, and giving me a precise way to quantify it.
You can't claim an organism gains information or loses information if you can't even define information or measure it, now can you?
While you're at it, tell me what you know about nylonase.
-------------- "CO2 can't re-emit any trapped heat unless all the molecules point the right way" "All the evidence supports Creation baraminology" "If it required a mind, planning and design, it isn't materialistic." "Jews and Christians are Muslims."
- Joke "Sharon" Gallien, world's dumbest YEC.
|