Utunumsint
Posts: 103 Joined: Jan. 2010
|
Quote (oldmanintheskydidntdoit @ Jan. 28 2010,16:10) | Quote (Utunumsint @ Jan. 28 2010,15:51) | 1-Has Lenski's results met these criteria? 2-Are the criteria themselves reasonable?
Cheers, Ut |
Behe: Quote | Yes, I’m perfectly willing to concede that this does appear to be the development of a new viral protein-viral protein binding site, one which I overlooked when writing about HIV. So the square point in Figure 7.4 representing HIV should be placed on the Y axis at a value of one, instead of zero, and Table 7.1 should list one protein-binding site developed by HIV instead of zero. |
How many more did he "overlook"? So, even by his own words his criteria must be falsified. He's no expert in the field. Miss one, miss 100. Same difference when you don't allow comments on your books at amazon, after all it's only about separating $$ from the faithful.
Ut-I'm not sure I understand the significance of this.... Can you dumb it down for me?
Quote | 2-Only cellular proteins binding to other cellular proteins are considered in this (viruses and other pathogens routinely bind to proteins, but do not create anything new, they only destroy what is already there). |
So Quote | 2-Are the criteria themselves reasonable? |
Perhaps. Define "destroy". Show that what happens when citrate becomes digestible is "destructive"? How? What was destroyed? How did you know the thing that was "destroyed" was not also "destroyed" itself previously? etc.
Oh, what's that? You could put the citrate digesting strain back into the original environment and see if it's beaten out by the "undamaged" bacteria you say? :p
Ut-I think when he made those comments, he was looking at the results of what he called the Trench warfare between malaria and the human immune system. Basically the human immune system was not able to combat malaria. It took a negavite mutation, like sickle cell anemia to provide some measure of defence. But it did so at a dreadful cost to the functionality of hemaglobin. He also provides other examples of mutations involving the hemaglobin, showing the same results. No new evolutionary function over this very prolongued conflict with malaria has evolved on the human side....
Anyway, I don't know if the above helps to clarify things.... what do you think?
Cheers, Ut |
Quote (Utunumsint @ Jan. 28 2010,15:51) | 1-Has Lenski's results met these criteria? 2-Are the criteria themselves reasonable?
Cheers, Ut |
Behe: Quote | Yes, I’m perfectly willing to concede that this does appear to be the development of a new viral protein-viral protein binding site, one which I overlooked when writing about HIV. So the square point in Figure 7.4 representing HIV should be placed on the Y axis at a value of one, instead of zero, and Table 7.1 should list one protein-binding site developed by HIV instead of zero. |
How many more did he "overlook"? So, even by his own words his criteria must be falsified. He's no expert in the field. Miss one, miss 100. Same difference when you don't allow comments on your books at amazon, after all it's only about separating $$ from the faithful.
Ut-I'm not sure I understand the significance of this.... Can you dumb it down for me?
Quote | 2-Only cellular proteins binding to other cellular proteins are considered in this (viruses and other pathogens routinely bind to proteins, but do not create anything new, they only destroy what is already there). |
So Quote | 2-Are the criteria themselves reasonable? |
Perhaps. Define "destroy". Show that what happens when citrate becomes digestible is "destructive"? How? What was destroyed? How did you know the thing that was "destroyed" was not also "destroyed" itself previously? etc.
Oh, what's that? You could put the citrate digesting strain back into the original environment and see if it's beaten out by the "undamaged" bacteria you say? :p
Ut-I think when he made those comments, he was looking at the results of what he called the Trench warfare between malaria and the human immune system. Basically the human immune system was not able to combat malaria. It took a negavite mutation, like sickle cell anemia to provide some measure of defence. But it did so at a dreadful cost to the functionality of hemaglobin. He also provides other examples of mutations involving the hemaglobin, showing the same results. No new evolutionary function over this very prolongued conflict with malaria has evolved on the human side....
Anyway, I don't know if the above helps to clarify things.... what do you think?
Cheers, Ut
|