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  Topic: Uncommonly Dense Thread 2, general discussion of Dembski's site< Next Oldest | Next Newest >  
KCdgw



Posts: 364
Joined: Sep. 2002

(Permalink) Posted: Aug. 25 2009,08:04   

Quote (Zachriel @ Aug. 25 2009,07:01)
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bornagain77: In fact, I followed this debate very closely and it turns out the trivial gain of just one protein-protein binding site being generated for the non-living HIV virus, that the evolutionists were “crowing” about, came at a staggering loss of complexity for the living host it invaded (People) with just that one trivial gain, (leaky cell membrane), in binding site complexity. Thus the “evolution” of the virus clearly stayed within the principle of Genetic Entropy since far more functional complexity was lost by the living human cells it invaded than ever was gained by the non-living HIV virus.

HIV is attempting to invade one of the most complex organisms on Earth, endowed not only with an elaborate immune system, but highly sophisticated technology. Yet, the virus is still capable of invading and feasting on human beings. From the point of view of the virus, a very important evolutionary advance.

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bornagain77: Thus the “evolution” of the virus clearly stayed within the principle of Genetic Entropy since far more functional complexity was lost by the living human cells it invaded than ever was gained by the non-living HIV virus.

Yes, that's the point. Just like the pouncing of a tiger, or the talons of a hawk.

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bornagain77: Do you have any actual empirical evidence that a fusion event has ever led to anything other than genetic diseases?

There are a lot of known examples. Among mammals, mice often change chromosome number as they evolve. Here's an interesting case.

Island mice may evolve faster: From one species to six

Britton-Davidian et al., Environmental genetics: Rapid chromosomal evolution in island mice, Nature 2000.

Update: "The extensive chromosomal divergence of the races in Madeira is expected to contribute to their genic divergence. However, there was no significant correlation between chromosomal and allozyme distances. This low apparent chromosomal impact on genic differentiation may be related to the short time since the onset of karyotypic divergence, as the strength of the chromosomal barrier will become significant only at later stages." (For students of speciation, that means chromosomal isolation before divergence.)

Britton-Davidian et al., Patterns of genic diversity and structure in a species undergoing rapid chromosomal radiation: an allozyme analysis of house mice from the Madeira archipelago, Nature 2007

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bornagain77: There is no logical “evolutionary” progression to be found for the amount of DNA in less complex animals to the DNA found in more complex animals.

That's because evolution isn't a progression, but a matter of happenstance with features cobbled together in response to immediate necessities, not due to some overarching plan.

I have pointed them to my series of essays on Human Chromsome 2 on PT.

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Those who know the truth are not equal to those who love it-- Confucius

  
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